E-Mail Edition Volume 12 Number 4
Published Fall, 2015
Published by Piccadilly Books, Ltd., www.piccadillybooks.com.
Bruce Fife, N.D., Publisher, www.coconutresearchcenter.org
If you would like to subscribe to the
Healthy Ways Newsletter
Contents:
-
Is There A Cure for Glaucoma?
-
Is A New Diet Pill Made from Coconut On the Horizon?
-
The Secret Formula for McDonald's French Fries
-
Are We Being Told the Truth About Vaccines?
Is There a Cure for Glaucoma?
A New Coconut Oil-Based Therapy Offers Promise
Joe Lovett was floored when he received
the news from his doctor—glaucoma. At that point his world was
shaken. Glaucoma is a degenerative eye disease characterized by
the gradual loss of peripheral vision that can progress to
tunnel vision and eventually complete blindness.
Joe was a
successful director and filmmaker living in New York City. In
the 1980s he was the producer for ABC News 20/20 program with
Hugh Downs and Barbara Walters. After leaving ABC News in 1989,
he founded his own company, Lovett Productions, producing
documentaries highlighting social and health issues.
In his early 20s Joe's eye doctor told
him his intraocular eye pressure was slightly elevated but that
he did not have to worry. So he didn't. Intraocular pressure is
the pressure of the fluid inside the eye. It wasn't until he was
in his early 40s that another doctor expressed concern about the
abnormally high eye pressure and sent him to an eye
specialist—an ophthalmologist—who could give him a definite
diagnoses. Joe was told he had glaucoma.
Elevated fluid pressure inside the eye
is the most characteristic feature of glaucoma and is the first
sign of the disease. It is believed that the excessive
intraocular pressure pushing on the retina and optic nerve at
the back of the eye causes them to degenerate. There is no cure
for glaucoma and standard treatment focuses on reducing eye
pressure to slow down and hopefully stop the progression of the
disease. Joe was given medicated eye
drops to help keep his eye pressure under control.
"I started in the traditional way with
eye drops but didn't take it too seriously," says Joe. "At that
time, I thought glaucoma meant that if you use the eye drops
you'll be fine. Not necessarily!"
Although tests indicated the he was
seeing well enough straight ahead, his side or peripheral vision
was degenerating and his vision was getting worse.
"One evening I was out walking when a
man suddenly appeared in front of me. It was, as they call it, a
classic 'jack-in-the-box' moment: one minute my view was clear
and the next minute, he popped suddenly into the middle of my
field of view." Joe shared this experience with his doctor and
who immediately referred him to a vision therapist.
"The vision therapist had me stand
across the room from her and asked me to focus one eye on her
nose and tell her how much of her body I could see. I couldn't
believe what I saw. She only existed above the shoulders and
below the hips. Her torso wasn't there. I literally felt sick.
It was the first time the reality sunk in."
The therapist explained that the
unconscious scanning of his eyes and visual memory probably kept
him from noticing the blind spots. She showed him where the
holes in his vision were and taught him, for safety reasons, how
to consciously scan his entire field of vision and bend his neck
and look down at every doorway. "I was devastated," Joe said.
The blind spots explained why he often
tripped when walking through a doorway or up dimly lit stairs.
He is still able to get around reasonable well by himself. He
can read, but he can only keep three to five words in focus at a
time. It's a struggle.
Despite medications and surgeries to
reduce the eye pressure, Joe has lost a third of his visual
field. "Supposing I lose all my vision," Joe wonders, "what do I
do?"
Losing your eyesight can be
devastating. We depend on sight for so many things in our lives,
that to be without it can be horrifying. Nearly half of all
adults worry more about vision loss than about losing their
memory or their ability to walk or hear. Among the elderly,
vision loss is their greatest fear next to death.
Glaucoma is the second most common
cause of blindness worldwide (cataracts is the first). It is
estimated that 1 out of every 120 people have glaucoma. Risk
increases with age.
Glaucoma is often called the "sneak
thief of sight" because it creeps up without warning and by the
time it is detected, substantial vision may already be lost.
Since there are few symptoms in the early stages, many people
have the disease without knowing it. It is estimated that as
many as half of those with glaucoma are not even aware of it.
Symptoms are subtle, however, there may be some hazy vision and
mild discomfort in the eyes and later a barely noticeable loss
of peripheral vision. As the disease progresses there is reduced
visual acuity and greatly increased fluid pressure that can
cause the appearance of colored rings or halos around bright
objects. Glaucoma can develop in one or both eyes.
There is no medically recognized cure
for glaucoma. Treatment focuses primarily on reducing pressure
and may include medicines, laser therapy, surgery, or a
combination of any of these. While these treatments may save
remaining vision, they do not improve sight already lost from
glaucoma. This is why early diagnosis is very important.
Anybody can develop glaucoma. We are
all at risk. While elevated fluid pressure in the eye is often
associated with the disease, the actual cause is unknown.
Lowering fluid eye pressure can slow down the progression of the
disease, yet in some cases it doesn't. Some people who develop
glaucoma have normal eye pressure and lowering pressure has not
proven to be beneficial. Elevated pressure may be a symptom
rather than the initiating factor.
When light enters the eye it is focused on the retina—the light sensitive nerve cells lining the back of the eyeball. The retina transforms light energy into a nerve impulse and sends the signal through the optic nerve to the brain where visual images are processed and interpreted. The eyes are actually a part of the brain. During fetal development, the brain pooches out to form the eyes.
Many researchers now view glaucoma as a neurologic disorder that causes nerve
Many researchers now view glaucoma as a neurologic disorder that causes nerve cells in the brain to degenerate and die, similar to what occurs in Parkinson's disease or Alzheimer's.
cells in
the brain to degenerate and die, similar to what occurs in
Parkinson's disease or Alzheimer's.1 Chronic
inflammation, characteristic of all neurodegenerative disorders,
is now becoming recognized as an important factor in glaucoma.
In Parkinson's disease the area of the brain called the
substantia nigra, which controls
movement, is affected most. In Alzheimer's it is the hippocampus
and frontal lobes, areas which involve memory. In glaucoma it is
the eyes.
Damage to,
and death of cells in the retina, mimics the same type of
degeneration in brain cells.
In fact, the same type of plaque that forms in the Alzheimer's
brain, also forms in the retina. Studies show that Alzheimer's
patients have an increased risk of developing glaucoma. For
example, a German study of institutionalized Alzheimer's
patients showed a 24.5 percent increased prevalence of glaucoma
compared to only 6.5 percent of age-matched patients without the
disease.2 A
Japanese study showed similar results. Alzheimer's patients had
an increased prevalence of glaucoma of 23.8 percent compared to
control patients with 9.9 percent.3 Not only are
Alzheimer's patients more likely to develop glaucoma, but
glaucoma patients are at increased risk of developing
Alzheimer's. A study of 812 glaucoma subjects, 72 years of age
and older, found that they were four times more likely to
develop dementia.4
While
intraocular pressure is an important diagnostic tool and
reducing abnormal pressure is still the standard form of
treatment, researchers are looking at new approaches. One of
these approaches is to take advantage of the body's own healing
mechanisms such as brain-derived neurotrophic factors (BDNFs).
Damage to, and death of cells in the retina, mimics the same type of degeneration in brain cells. In fact, the same type of plaque that forms in the Alzheimer's brain, also forms in the retina.
They protect neurons from the
detrimental effects of various toxins and stressors that can
harm brain and nerve tissue.5-6 BDNFs help to support
the survival of existing neurons and encourage the growth and
differentiation of new neurons. BDNFs play a significant role in
the maintenance of brain and eye cell function throughout an
individual's lifetime.
Initial studies using rats with
glaucoma have shown that when BDNFs are injected into the eyes,
retinal cell degeneration and loss abruptly declines.7
However, the effect is only temporary. Multiple injections are
necessary for significant beneficial effect, reducing the
clinical usefulness of this approach. A continuous supply of
BDNFs is needed in order to have a beneficial outcome. While
injecting BDNFs may not be practical, raising and sustaining
blood levels of BDNFs naturally is very doable. The key is to
boost blood ketone levels.
The brain uses glucose as its primary
source of fuel. Whenever we eat a meal, much of the food is
converted into glucose and released into the bloodstream.
However, if we don't eat for several hours or we don't eat any
foods that can be converted into glucose (primarily
carbohydrates), blood glucose levels begin to fall, as they do,
the liver starts producing ketones from stored fat. Ketones act
as an alternative source of fuel for the brain. If you eat two
or three meals a day, blood ketone levels are normally kept
fairly low. Blood ketones can be raised by prolonged fasting or
by eating a very low-carb or ketogenic diet.
Ketones are known to stimulate the
production of BDNFs. Consuming a ketogenic diet can raise BDNFs
to levels that can have therapeutic effects on the brain and the
eyes.
Another way to raise blood
ketones is by eating coconut oil. When coconut oil is consumed,
a portion of the medium chain fatty acids (MCFAs) in the oil
will automatically be converted into ketones, regardless of
blood glucose levels.8
You can raise blood ketones, and subsequently BDNFs, to
therapeutic levels by eating coconut oil.
Consuming a ketogenic diet can raise BDNFs to levels that can have therapeutic effects on the brain and the eyes.
production of
ketones and BDNFs, improving their therapeutic effect. If a
ketogenic coconut oil-based diet is maintained for a period of
time, it can allow the time needed to bring about healing and
repair within the central nervous system. Ketones have proven to
be successful in reversing Epilepsy, Alzheimer's, Parkinson's
and other neurodegenerative diseases.9-12
The eyes are also part of the central
nervous system and can be protected with ketone-induced BDNFs.
In animal studies where the retina and optic nerve are
intentionally injured, BDNFs have proven to lessen the damage
and stimulate healing and regrowth of these tissues, preserving
eyesight.13 This has some very pronounced
ramifications. It means that, despite what you may have been
told, degenerative eye diseases like glaucoma are not
necessarily irreversible.
I was able to reverse my own glaucoma using coconut oil. Eighteen years ago I was diagnosed with glaucoma. I didn't go on medication but changed my diet and started using coconut oil in place of other vegetable oils.
I reversed my own glaucoma using coconut oil.
Two years later I was pronounced
glaucoma-free. I've been tested every few years since and still
no sign of glaucoma or loss of vision. In fact, my vision has
improved since the initial diagnosis. The details of my story
are in the book
Stop Vision Loss Now!: Prevent and Heal Cataracts, Glaucoma,
Macular Degeneration, and Other Common Eye Disorders.14
treatment.
An easy way to help prevent vision loss is to add coconut oil
into your daily diet. I recommend adding 1-3
tablespoons of coconut oil
daily to your meals. You can use the oil in cooking and baking
or add it on afterwards. There is more to preserving and
restoring eyesight than just eating coconut oil. A healthy diet
is also essential along with avoiding troublemakers like tobacco
smoke, certain drugs and food additives, and excessive sugar and
refined carbohydrates.
To find out more about Dr. Bruce Fife's newest book, Stop Vision Loss Now!, or to order click here.
References
1. Chang,
EE and Goldberg, JL. Glaucoma 2.0: neuroprotection,
neuroregeneration, neuroenhancement..
Ophthalmology
2012;119:979-986.
2. Bayer AU, Ferrari F, Erb
C. High occurrence rate of glaucoma among patients with
Alzheimer's disease.
Eur Neurol.
2002;47(3):165-168.
3. Tamura
H, Kawakami H, Kanamoto T, et al. High frequency of open-angle
glaucoma in Japanese patients with Alzheimer's disease.
J Neurol Sci. Jul
15 2006;246(1-2):79-83.
4. Helmer
C, Malet F, Rougier M-B, et al. Is there a link between
open-angle glaucoma and dementia?: The Three-City—Alienor Cohort.
Ann Neurol.
2013;74:171-179
5. Duan, W.
and Mattson, M.P. Dietary restriction and 2-deoxyglucose
administration improve behavioral outcome and reduce
degeneration of dopaminergic neurons in models of Parkinson's
disease. J Neurosci Res
1999;57:195-206.
6. Mattson,
M.P. Neuroprotective signaling and the aging brain: take away my
food and let me run.
Brain Res
2000;886:47-53.
7. Ko, ML,
et al. Patterns of retinal ganglion cell survival after
brain-derived neurotrophic factor administration in hypertensive
eyes of rats.
Neurosci Lett
2001:305:139-142.
8. Bergen,
SS Jr., et al. Hyperketonemia induced in man by medium-chain
triglyceride.
Diabetes
1966;15:723-725.
9. Liu, YM
and Wang, HS. Medium-chain triglyceride ketogenic diet, an
effective treatment for drug-resistant epilepsy and a comparison
with other ketogenic diets.
Biomed J
2013;36:9-15.
10.
VanItallie, TB, et al. Treatment of Parkinson disease with
diet-induced hyperketonemia: a feasibility study.
Neurology
2005;64:728-730.
11. Reger,
MA, et al. Effects of beta-hydroxybutyrate on cognition in
memory-impaired adults.
Neurobiol Aging
2004;25:311-314.
12. Fife,
B.,
Stop Alzheimer's Now!: How to
Prevent and Reverse Dementia, Parkinson's, ALS, Multiple
Sclerosis, and Other Neurodegenerative Disorders.
Piccadilly Books,
Ltd. 2011.
13. Weibel,
D, et al. Brain-derived neurotrophic factor (BDNF) prevents
lesion-induced axonal die-back in young rat optic nerve.
Brain Res
1995;679:249-254.
14. Fife,
B.
Stop Vision Loss Now!: Prevent and
Heal Cataracts, Glaucoma, Macular Degeneration, and Other Common
Eye Disorders; Piccadilly
Books, Ltd. 2015.
Is A New Diet Pill Made from Coconut On the Horizon?
Scientists say it could reduce calorie intake by 18 percent
Are you overweight? Tried dieting without success? One of, if not the hardest challenge with dieting is enduring the constant nagging hunger. What if there was a pill you could take that could stop hunger, or at least significantly reduce it? You feel satisfied eating less food and
consuming
fewer calories. That would make dieting far more comfortable and
increase a person's chances of success. Well, researchers in Australia
think they have discovered the makings for
such a pill and it comes from the oddest source — coconut.
This new appetite-suppressing obesity pill is
currently under development by researchers at the University of
Adelaide. Preliminary studies have shown it to reduce calorie intake by
up to 18 percent. The researchers discovered that feeding human subjects
concentrated amounts of lauric acid reduced their appetite to the point
where they consumed 10-18 percent fewer calories.1
Lauric acid is the primary fatty acid found in
coconut oil, making up to 50 percent of the oil. Lead researcher
Christine Feinle-Bisset, PhD, says she's working with a biotech company
to commercialize the discovery and says a pill is "not too far off."
However, she was quick to add, "The purpose of our research is not to
advise people to include coconut oil in their diet." Her team used an
extract of coconut oil and she says they did not look at what would
happen if the whole oil was given to people. Interesting comment since
people have been eating coconut oil for thousands of years without any
apparent harm.
Perhaps her concern is that if people started
using ordinary, inexpensive coconut oil, there would be no need to
purchase costly diet pills—the ones she would be involved in producing.
So it is understandable why she isn't too keen on recommending coconut
oil over her new discovery.
The discovery emerged from government funded
research Dr. Feinle-Bisset undertook to explore how certain nutrients
can affect satiety censors in the intestine. "Some years ago we found
particular nutrients in small amounts have a potent effect to suppress
nutrient intake," she said.
Subjects in her study were fed lauric acid via
a tube inserted through their nose that went straight to their small
intestine. Their energy intake was compared to a control group who were
given a salt infusion. "In the end, there was a 10-18 percent reduction
in calories with no side effects," she said.
In the initial study, some patients experienced
nausea, so the research team experimented with lower doses and found a
dose that was tolerated without side effects. The amount just happened
to be small enough to fit into a standard dietary capsule. Later studies
using the capsules administered orally in the morning found it reduced
the amount of food and total calorie intake by test subjects when they
ate lunch.
Australia's Health Minister, Sussan Ley, says
this breakthrough was one of the ten groundbreaking medical studies
funded by the government along with a new technique to train the immune
system to identify and attack cancer cells and a new device that can
detect and stop epileptic seizures as they happen.
Dr. Feinle-Bisset has won a new National Health
and Medical Research Council grant and plans to examine how other
nutrients including amino acids in high protein diets help suppress
appetite.
The fact that
lauric acids helps reduce hunger is not really a new discovery. Studies
as far back as 1996 have shown that medium chain fatty acids from
coconut oil produce greater satiety than fats from common vegetable
oils.2
A slew of studies over the years have shown
that diets that contain coconut oil promote greater weight loss than
diets using other oils. This is not due just to the satiety factor but
to other effects such as its ability to boost metabolism and turn off
enzymes that convert calories into fat.3
A new study published earlier this year
provided more evidence for coconut oil's growing reputation as a weight
management aid. The study compared the health effects of coconut oil
with soybean oil. In this study, scientists divided the mice into four
groups and fed them each a high-calorie, high-fat diet to promote weight
gain. Each of the four diets contained 40 percent fat. One diet used
coconut oil, in another group half of the coconut oil was replaced with
soybean oil (which primarily contains polyunsaturated fat). The third
and fourth diets were the same as the first two but had fructose added.
All four diets had the same number of calories, and the mice were fed
the same amount of food.
As expected, all of the mice gained weight on
the high-calorie diet, but at distinctly different rates. The mice that
were on the coconut oil diet gained the least amount, a whopping 25
percent less weight than those on the soybean oil diet. Interestingly,
the mice on the soybean oil diet also gained 12 percent more weight than
those that ate a fructose diet.
The mice on the soybean oil diet also had
larger fat deposits in their bodies, developed fatty livers, and were
more likely to develop diabetes and insulin resistance. Also, genes
involved mitochondrial dysfunction and cancer were upregulated by the
soybean oil diet. Mice on the fructose diet didn't get off easy, either
— they had similar issues, but to a less severe degree.4
This study provided further evidence that using
coconut oil in place of polyunsaturated vegetable oils, such as soybean
oil, can help you better manage your weight and reduce the risks of
fatty liver, insulin resistance, diabetes, and even cancer.
And, it's worth noting, the amount of soybean
oil the mice ate was similar to what we typically get in our diets.
The bottom line is that we don't need a
commercially produced coconut oil-based diet pill to help us manage our
weight. Simply replacing the polyunsaturated vegetables oils in our diet
with coconut oil can help us better manage our weight and help reduce
our risks of liver disease, diabetes, and cancer.
References
1. Feltrin, KL, et al. Effects of lauric acid
on upper gut motility, plasma cholecystokinin and peptide YY, and energy
intake are load, but not concentration, dependent in humans. Journal of
Physiology 2007;581;767-777.
2. Stubbs, RJ and Harbron, CG. Covert
manipulation of the ratio of medium-to long-chain triglycerides in
isoenergetically dense diets: effect on food intake in ad libitum
feeding men. Int J Obes Relat Metab Disord 1996;20:435-444.
3. Fife, B. The Coconut Ketogenic Diet:
Supercharge Your Metabolism, Revitalize Thyroid Function, and Lose
Excess Weight. Piccadilly Books, Ltd. 2014.
4. Deol, P, et al. Soybean oil is more
obesogenic and diabetogenic than coconut oil and fructose in mouse:
potential role for the liver. PLoS ONE 20015;10(7):e0132672.
The Secret Formula for McDonald's French Fries
Does it even contain any real potatoes?You might be surprised
Ever since
McDonald's first opened in 1948, the one thing most people rave about is
their French fries. As a kid, I loved their French fries better than any
other and would go there just for their fries.
The original fries were made from russet
potatoes, cooked in beef tallow, and finished off with a little salt.
Simple, but delicious, primarily because they were cooked in beef tallow
that gave the fries their unique savory flavor. However, with the outcry
against saturated fats in the late 1980s, most restaurants, including
McDonald's, in order to ease customer's fears, replaced the beef tallow
with hydrogenated soybean oil. Ignoring the fact that hydrogenated
vegetable oils are far worst for your health than beef tallow, the
change in oils made a huge impact on the taste and flavor of the fries.
Hydrogenated soybean oil, which is highly
processed and tasteless, produced a bland, flavorless French fry that
had no resemblance to the delicious fries cooked in beef tallow. So
McDonald's began experimenting with the formula adding beef broth and
flavorings to mimic the flavor and texture of their original fries. Even
the types of oils they used changed over the years. So many changes were
made that some people even wondered if they even contained any real
potatoes.
Food myth buster, Grant Imahara, was allowed
access into the McDonald's French fry processing plant in Idaho, to set
the record straight about what ingredients go into McDonald's secret
French fry formula.
After visiting the plant and observing the
entire process involved in producing the fries, he was able to confirm
that potatoes really are the first and primary ingredient in their
fries. But don't get too excited just yet. In addition to the potatoes,
there are another 18 ingredients! That's right. A total of 19
ingredients goes into making that simple little French fry. And some of
these ingredients are not what you might expect.
The formula includes 14 chemicals, one of which
is petroleum-based (tertiary butylhydroquinone) and another is
a silicone used in silly putty (dimethylpolysiloxane). The fries are
made from a mix of russet, Burbank, ranger russet, Umatilla russet, and
Shepody potatoes then are peeled cut and blanched before being fired
through a cutting cannon at 70 mph. The cut potatoes then pass through a
bath of sauces, consisting of a blend of canola oil, corn oil, soybean
oil, hydrogenated soybean oil, natural beef flavor, hydrolysed wheat,
hydrolysed milk, citric acid, Tertiary butylhydroquinone (TBHQ), and
dimethylpolysiloxane. TBHQ is included in the cooking oil as a food
preservative and dimethylpolysiloxane is used to stop cooking oils from
foaming as the potatoes are deep fried. Finally, dextrose is then
sprayed on the fries to ensure they maintain a golden color and sodium
acid pyrophosphate is added to stop them from turning gray.
After the potatoes are fried in the factory,
they are then flash frozen and shipped off to all the McDonald's
restaurants, where they are fried a second time and salted just before
serving. The end product is mmmm, mmmm, finger lick'in good...or is that
some other fried product with mystery ingredients?
♦
Are We Being Told the Truth About
Vaccines?
Whistleblower Reveals CDC Destroyed Documents to Conceal Cover Up of MMR-Autism Link
On July 29, 2015,
US Congressman Bill Posey spoke on the floor of the US House quoting
whistleblower Dr. Bill Thompson, a senior research scientist at the
Center for Disease Control and Prevention (CDC). Dr. Thompson is quoted
as stating that the CDC purposely destroyed documents to conceal their
tracks when they covered up their own study's findings that MMR vaccine
caused a huge risk of autism in black boys when given before 3 years of
age.
The study was published in the
Journal of Pediatrics and was
used by the pro-vaccine lobby as "proof" that the MMR vaccine did not
cause autism. It also provided ammunition to push new vaccine
legislation though state governments and restrict parents' rights to
make informed decision regarding vaccinations for their children. For
example, California's recent move to eliminate all personal exceptions
for children from receiving the standard 48 or so vaccinations by age 6
currently mandated.
Below is an excerpt of Congressman Posey's address:
That being said, it's troubling to me that in a
recent Senate hearing on childhood vaccinations, it was never mentioned
that our government has paid out over three billion dollars through a
vaccine injury compensation program for children who have been injured
by vaccinations. Regardless of the subject matter, parents making
decisions about their children's health deserve to have the best
information available to them. They should be able to count on federal
agencies to tell them the truth. For these reasons I bring the following
matter to the House floor.
In August 2014, Dr. William Thompson, a senior
scientist at the Centers for Disease Control and Prevention worked with
a whistleblower attorney to provide my office with documents related to
a 2004 CDC study that examined the possibility of a relationship between
mumps, measles, rubella (MMR) vaccines and autism. In a statement
released in August 2014, Dr. Thompson stated, "I regret that my
co-authors and I omitted," OMITTED, "statistically significant
information in our 2004 article published in the
Journal of Pediatrics."
Mr. Speaker, I respectfully request the
following excerpts from the statement written by Dr. Thompson be entered
into the record. Now quoting Dr. Thompson:
"My primary job duties while working in the
immunization safety branch from 2000-06 were to lead or co-lead three
major vaccine safety studies. The MADDSP MMR Autism Cases controlled
study was being carried out in response to the Wakefield
Lancet study that suggested an association between the MMR vaccine
and an autism-like health outcome. There were several major concerns
among scientists and consumers advocates outside the CDC in the fall of
2000 regarding the execution of the Verstraeten study. One of the
important goals that was determined up front in the spring of '01 before
any of these studies started was to have all three protocols vetted
outside the CDC prior to start of the analyses so that consumer
advocates could not claim that we were presenting analyses that suited
our own goals and biases. We hypothesized that if we found statistically
significant effects at either 18 or 36 month thresholds, we would
conclude that vaccinating children early with MMR vaccine could lead to
autism-like characteristics or features.
"We all met and finalized the study protocol
and analysis plan. The goal was to not deviate from the analysis plan to
avoid the debacle that occurred with the Verstraeten thimerosal study
published in Pediatrics in
'03. At the September 5th meeting we discussed in detail how to code
race for both the sample and the birth certificate sample. At the bottom
of Table 7, it also shows that for the non-birth-certificate sample, the
adjusted race effect statistical significance was huge. All the authors
and I met and decided sometime between August and September '02 not to
report any race effects for the paper. Sometime soon after the meeting
we decided to exclude reporting any race effects, the co-authors
scheduled a meeting to destroy documents related to the study. The
remaining four co-authors all met and brought a big garbage can into the
meeting room and reviewed and went through all the hardcopy documents
that we had thought we should discard and put them in a huge garbage
can. However, because I assumed it was illegal and would violate both
FOIA and DOJ requests, I kept hard copies of all documents in my office,
and I retained all associated computer files.
"I believe we intentionally withheld
controversial findings from the final draft of the Pediatrics paper."
Mr. Speaker, I believe it's our duty to insure
that the documents Dr. Thompson provided are not ignored. Therefore, I
will provide them to members of Congress and the House committees upon
request. Considering the nature of the whistleblower's documents, as
well as the involvement of the CDC, a hearing and a thorough
investigation is warranted. So I ask, Mr. Speaker, I beg, I implore my
colleagues on the appropriations committees to please, please take such
action. Thank you, Mr. Speaker.
The vaccine makers and their allies in
government, are working to take away our right to be informed about the
dangers of vaccinations and to force us to be vaccinated, even against
our will. If this is of concern to you, I urge you to take action and
let your voice be heard.
Send an email to Congressman Posey AND your own
Congressperson. The email does not have to be long, all it needs
to say is something like this:
"I support Rep. Bill Posey and thank him for
his testimony on July 29, 2015 regarding the CDC's intentional
destruction of documents. I am grateful William Thompson, MD retained
hard copies documenting the truth and as a US citizen I am requesting a
full investigation into this issue."
Remember to include your name and address.
1) PLEASE EMAIL CONGRESSMAN POSEY:
Send EMAILS of support directly to Bill Posey's
assistant Patricia Febro at Patricia.Febro@mail.house.gov.
2) PLEASE ALSO EMAIL or CALL YOUR OWN
CONGRESSPERSON.
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Copyright © 2015, Bruce Fife. All rights reserved.