E-Mail Edition  Volume 12   Number 4

Published Fall, 2015

Published by Piccadilly Books, Ltd., www.piccadillybooks.com.

Bruce Fife, N.D., Publisher, www.coconutresearchcenter.org  

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  • Is There A Cure for Glaucoma?

  • Is A New Diet Pill Made from Coconut On the Horizon?

  • The Secret Formula for McDonald's French Fries

  • Are We Being Told the Truth About Vaccines?


Is There a Cure for Glaucoma?

A New Coconut Oil-Based Therapy Offers Promise


Joe Lovett was floored when he received the news from his doctor—glaucoma. At that point his world was shaken. Glaucoma is a degenerative eye disease characterized by the gradual loss of peripheral vision that can progress to tunnel vision and eventually complete blindness.

Tunnel-vision view from glaucoma

Joe was a successful director and filmmaker living in New York City. In the 1980s he was the producer for ABC News 20/20 program with Hugh Downs and Barbara Walters. After leaving ABC News in 1989, he founded his own company, Lovett Productions, producing documentaries highlighting social and health issues.

In his early 20s Joe's eye doctor told him his intraocular eye pressure was slightly elevated but that he did not have to worry. So he didn't. Intraocular pressure is the pressure of the fluid inside the eye. It wasn't until he was in his early 40s that another doctor expressed concern about the abnormally high eye pressure and sent him to an eye specialist—an ophthalmologist—who could give him a definite diagnoses. Joe was told he had glaucoma.

Elevated fluid pressure inside the eye is the most characteristic feature of glaucoma and is the first sign of the disease. It is believed that the excessive intraocular pressure pushing on the retina and optic nerve at the back of the eye causes them to degenerate. There is no cure for glaucoma and standard treatment focuses on reducing eye pressure to slow down and hopefully stop the progression of the disease. Joe was given medicated eye drops to help keep his eye pressure under control.

"I started in the traditional way with eye drops but didn't take it too seriously," says Joe. "At that time, I thought glaucoma meant that if you use the eye drops you'll be fine. Not necessarily!"

Although tests indicated the he was seeing well enough straight ahead, his side or peripheral vision was degenerating and his vision was getting worse.

"One evening I was out walking when a man suddenly appeared in front of me. It was, as they call it, a classic 'jack-in-the-box' moment: one minute my view was clear and the next minute, he popped suddenly into the middle of my field of view." Joe shared this experience with his doctor and who immediately referred him to a vision therapist.

"The vision therapist had me stand across the room from her and asked me to focus one eye on her nose and tell her how much of her body I could see. I couldn't believe what I saw. She only existed above the shoulders and below the hips. Her torso wasn't there. I literally felt sick. It was the first time the reality sunk in."

The therapist explained that the unconscious scanning of his eyes and visual memory probably kept him from noticing the blind spots. She showed him where the holes in his vision were and taught him, for safety reasons, how to consciously scan his entire field of vision and bend his neck and look down at every doorway. "I was devastated," Joe said.

The blind spots explained why he often tripped when walking through a doorway or up dimly lit stairs. He is still able to get around reasonable well by himself. He can read, but he can only keep three to five words in focus at a time. It's a struggle.

Despite medications and surgeries to reduce the eye pressure, Joe has lost a third of his visual field. "Supposing I lose all my vision," Joe wonders, "what do I do?"

Losing your eyesight can be devastating. We depend on sight for so many things in our lives, that to be without it can be horrifying. Nearly half of all adults worry more about vision loss than about losing their memory or their ability to walk or hear. Among the elderly, vision loss is their greatest fear next to death.

Glaucoma is the second most common cause of blindness worldwide (cataracts is the first). It is estimated that 1 out of every 120 people have glaucoma. Risk increases with age.

Glaucoma is often called the "sneak thief of sight" because it creeps up without warning and by the time it is detected, substantial vision may already be lost. Since there are few symptoms in the early stages, many people have the disease without knowing it. It is estimated that as many as half of those with glaucoma are not even aware of it. Symptoms are subtle, however, there may be some hazy vision and mild discomfort in the eyes and later a barely noticeable loss of peripheral vision. As the disease progresses there is reduced visual acuity and greatly increased fluid pressure that can cause the appearance of colored rings or halos around bright objects. Glaucoma can develop in one or both eyes.

There is no medically recognized cure for glaucoma. Treatment focuses primarily on reducing pressure and may include medicines, laser therapy, surgery, or a combination of any of these. While these treatments may save remaining vision, they do not improve sight already lost from glaucoma. This is why early diagnosis is very important.

Anybody can develop glaucoma. We are all at risk. While elevated fluid pressure in the eye is often associated with the disease, the actual cause is unknown. Lowering fluid eye pressure can slow down the progression of the disease, yet in some cases it doesn't. Some people who develop glaucoma have normal eye pressure and lowering pressure has not proven to be beneficial. Elevated pressure may be a symptom rather than the initiating factor.


When light enters the eye it is focused on the retina—the light sensitive nerve cells lining the back of the eyeball. The retina transforms light energy into a nerve impulse and sends the signal through the optic nerve to the brain where visual images are processed and interpreted. The eyes are actually a part of the brain. During fetal development, the brain pooches out to form the eyes.

Many researchers now view glaucoma as a neurologic disorder that causes nerve

Many researchers now view glaucoma as a neurologic disorder that causes nerve cells in the brain to degenerate and die, similar to what occurs in Parkinson's disease or Alzheimer's.

cells in the brain to degenerate and die, similar to what occurs in Parkinson's disease or Alzheimer's.1 Chronic inflammation, characteristic of all neurodegenerative disorders, is now becoming recognized as an important factor in glaucoma. In Parkinson's disease the area of the brain called the substantia nigra, which controls movement, is affected most. In Alzheimer's it is the hippocampus and frontal lobes, areas which involve memory. In glaucoma it is the eyes.

Damage to, and death of cells in the retina, mimics the same type of degeneration in brain cells. In fact, the same type of plaque that forms in the Alzheimer's brain, also forms in the retina. Studies show that Alzheimer's patients have an increased risk of developing glaucoma. For example, a German study of institutionalized Alzheimer's patients showed a 24.5 percent increased prevalence of glaucoma compared to only 6.5 percent of age-matched patients without the disease.2 A Japanese study showed similar results. Alzheimer's patients had an increased prevalence of glaucoma of 23.8 percent compared to control patients with 9.9 percent.3 Not only are Alzheimer's patients more likely to develop glaucoma, but glaucoma patients are at increased risk of developing Alzheimer's. A study of 812 glaucoma subjects, 72 years of age and older, found that they were four times more likely to develop dementia.4

While intraocular pressure is an important diagnostic tool and reducing abnormal pressure is still the standard form of treatment, researchers are looking at new approaches. One of these approaches is to take advantage of the body's own healing mechanisms such as brain-derived neurotrophic factors (BDNFs).

BDNFs play a key role in regulating survival, growth, and maintenance of neurons. They regulate the neurotransmitters (e.g., dopamine, glutamate) that carry chemical signals, which

Damage to, and death of cells in the retina, mimics the same type of degeneration in brain cells. In fact, the same type of plaque that forms in the Alzheimer's brain, also forms in the retina.

allow neurons to communicate with each other.

They protect neurons from the detrimental effects of various toxins and stressors that can harm brain and nerve tissue.5-6 BDNFs help to support the survival of existing neurons and encourage the growth and differentiation of new neurons. BDNFs play a significant role in the maintenance of brain and eye cell function throughout an individual's lifetime.

Initial studies using rats with glaucoma have shown that when BDNFs are injected into the eyes, retinal cell degeneration and loss abruptly declines.7 However, the effect is only temporary. Multiple injections are necessary for significant beneficial effect, reducing the clinical usefulness of this approach. A continuous supply of BDNFs is needed in order to have a beneficial outcome. While injecting BDNFs may not be practical, raising and sustaining blood levels of BDNFs naturally is very doable. The key is to boost blood ketone levels.

The brain uses glucose as its primary source of fuel. Whenever we eat a meal, much of the food is converted into glucose and released into the bloodstream. However, if we don't eat for several hours or we don't eat any foods that can be converted into glucose (primarily carbohydrates), blood glucose levels begin to fall, as they do, the liver starts producing ketones from stored fat. Ketones act as an alternative source of fuel for the brain. If you eat two or three meals a day, blood ketone levels are normally kept fairly low. Blood ketones can be raised by prolonged fasting or by eating a very low-carb or ketogenic diet.

Ketones are known to stimulate the production of BDNFs. Consuming a ketogenic diet can raise BDNFs to levels that can have therapeutic effects on the brain and the eyes.

Another way to raise blood ketones is by eating coconut oil. When coconut oil is consumed, a portion of the medium chain fatty acids (MCFAs) in the oil will automatically be converted into ketones, regardless of blood glucose levels.8 You can raise blood ketones, and subsequently BDNFs, to therapeutic levels by eating coconut oil.  

Combining coconut oil with a ketogenic diet enhances the

Consuming a ketogenic diet can raise BDNFs to levels that can have therapeutic effects on the brain and the eyes.

production of ketones and BDNFs, improving their therapeutic effect. If a ketogenic coconut oil-based diet is maintained for a period of time, it can allow the time needed to bring about healing and repair within the central nervous system. Ketones have proven to be successful in reversing Epilepsy, Alzheimer's, Parkinson's and other neurodegenerative diseases.9-12

The eyes are also part of the central nervous system and can be protected with ketone-induced BDNFs. In animal studies where the retina and optic nerve are intentionally injured, BDNFs have proven to lessen the damage and stimulate healing and regrowth of these tissues, preserving eyesight.13 This has some very pronounced ramifications. It means that, despite what you may have been told, degenerative eye diseases like glaucoma are not necessarily irreversible.


I was able to reverse my own glaucoma using coconut oil. Eighteen years ago I was diagnosed with glaucoma. I didn't go on medication but changed my diet and started using coconut oil in place of other vegetable oils.

I reversed my own glaucoma using coconut oil.

 Two years later I was pronounced glaucoma-free. I've been tested every few years since and still no sign of glaucoma or loss of vision. In fact, my vision has improved since the initial diagnosis. The details of my story are in the book Stop Vision Loss Now!: Prevent and Heal Cataracts, Glaucoma, Macular Degeneration, and Other Common Eye Disorders.14 Of course, prevention is always much easier than

treatment. An easy way to help prevent vision loss is to add coconut oil into your daily diet. I recommend adding 1-3 tablespoons of coconut oil daily to your meals. You can use the oil in cooking and baking or add it on afterwards. There is more to preserving and restoring eyesight than just eating coconut oil. A healthy diet is also essential along with avoiding troublemakers like tobacco smoke, certain drugs and food additives, and excessive sugar and refined carbohydrates.


Stop Vision Loss Now by Dr. Bruce Fife ND

To find out more about Dr. Bruce Fife's newest book, Stop Vision Loss Now!, or to order click here.



1. Chang, EE and Goldberg, JL. Glaucoma 2.0: neuroprotection, neuroregeneration, neuroenhancement.. Ophthalmology 2012;119:979-986.

2. Bayer AU, Ferrari F, Erb C. High occurrence rate of glaucoma among patients with Alzheimer's disease. Eur Neurol. 2002;47(3):165-168.

3. Tamura H, Kawakami H, Kanamoto T, et al. High frequency of open-angle glaucoma in Japanese patients with Alzheimer's disease. J Neurol Sci. Jul 15 2006;246(1-2):79-83.

4. Helmer C, Malet F, Rougier M-B, et al. Is there a link between open-angle glaucoma and dementia?: The Three-City—Alienor Cohort. Ann Neurol. 2013;74:171-179

5. Duan, W. and Mattson, M.P. Dietary restriction and 2-deoxyglucose administration improve behavioral outcome and reduce degeneration of dopaminergic neurons in models of Parkinson's disease. J Neurosci Res 1999;57:195-206.

6. Mattson, M.P. Neuroprotective signaling and the aging brain: take away my food and let me run. Brain Res 2000;886:47-53.

7. Ko, ML, et al. Patterns of retinal ganglion cell survival after brain-derived neurotrophic factor administration in hypertensive eyes of rats. Neurosci Lett 2001:305:139-142.

8. Bergen, SS Jr., et al. Hyperketonemia induced in man by medium-chain triglyceride. Diabetes 1966;15:723-725.

9. Liu, YM and Wang, HS. Medium-chain triglyceride ketogenic diet, an effective treatment for drug-resistant epilepsy and a comparison with other ketogenic diets. Biomed J 2013;36:9-15.

10. VanItallie, TB, et al. Treatment of Parkinson disease with diet-induced hyperketonemia: a feasibility study. Neurology 2005;64:728-730.

11. Reger, MA, et al. Effects of beta-hydroxybutyrate on cognition in memory-impaired adults. Neurobiol Aging 2004;25:311-314.

12. Fife, B., Stop Alzheimer's Now!: How to Prevent and Reverse Dementia, Parkinson's, ALS, Multiple Sclerosis, and Other Neurodegenerative Disorders. Piccadilly Books,  Ltd. 2011.

13. Weibel, D, et al. Brain-derived neurotrophic factor (BDNF) prevents lesion-induced axonal die-back in young rat optic nerve. Brain Res 1995;679:249-254.

14. Fife, B. Stop Vision Loss Now!: Prevent and Heal Cataracts, Glaucoma, Macular Degeneration, and Other Common Eye Disorders; Piccadilly Books, Ltd. 2015.


Is A New Diet Pill Made from Coconut On the Horizon?

diet pill yellow gel caps

Scientists say it could reduce calorie intake by 18 percent

Are you overweight? Tried dieting without success? One of, if not the hardest challenge with dieting is enduring the constant nagging hunger. What if there was a pill you could take that could stop hunger, or at least significantly reduce it? You feel satisfied eating less food and

Man on a diet.

fat rat

consuming fewer calories. That would make dieting far more comfortable and increase a person's chances of success. Well, researchers in Australia think they have discovered the makings for such a pill and it comes from the oddest source — coconut.

This new appetite-suppressing obesity pill is currently under development by researchers at the University of Adelaide. Preliminary studies have shown it to reduce calorie intake by up to 18 percent. The researchers discovered that feeding human subjects concentrated amounts of lauric acid reduced their appetite to the point where they consumed 10-18 percent fewer calories.1

Lauric acid is the primary fatty acid found in coconut oil, making up to 50 percent of the oil. Lead researcher Christine Feinle-Bisset, PhD, says she's working with a biotech company to commercialize the discovery and says a pill is "not too far off." However, she was quick to add, "The purpose of our research is not to advise people to include coconut oil in their diet." Her team used an extract of coconut oil and she says they did not look at what would happen if the whole oil was given to people. Interesting comment since people have been eating coconut oil for thousands of years without any apparent harm.

Perhaps her concern is that if people started using ordinary, inexpensive coconut oil, there would be no need to purchase costly diet pills—the ones she would be involved in producing. So it is understandable why she isn't too keen on recommending coconut oil over her new discovery.

The discovery emerged from government funded research Dr. Feinle-Bisset undertook to explore how certain nutrients can affect satiety censors in the intestine. "Some years ago we found particular nutrients in small amounts have a potent effect to suppress nutrient intake," she said.

Subjects in her study were fed lauric acid via a tube inserted through their nose that went straight to their small intestine. Their energy intake was compared to a control group who were given a salt infusion. "In the end, there was a 10-18 percent reduction in calories with no side effects," she said.

In the initial study, some patients experienced nausea, so the research team experimented with lower doses and found a dose that was tolerated without side effects. The amount just happened to be small enough to fit into a standard dietary capsule. Later studies using the capsules administered orally in the morning found it reduced the amount of food and total calorie intake by test subjects when they ate lunch.

Australia's Health Minister, Sussan Ley, says this breakthrough was one of the ten groundbreaking medical studies funded by the government along with a new technique to train the immune system to identify and attack cancer cells and a new device that can detect and stop epileptic seizures as they happen.

Dr. Feinle-Bisset has won a new National Health and Medical Research Council grant and plans to examine how other nutrients including amino acids in high protein diets help suppress appetite.

The fact that lauric acids helps reduce hunger is not really a new discovery. Studies as far back as 1996 have shown that medium chain fatty acids from coconut oil produce greater satiety than fats from common vegetable oils.2

A slew of studies over the years have shown that diets that contain coconut oil promote greater weight loss than diets using other oils. This is not due just to the satiety factor but to other effects such as its ability to boost metabolism and turn off enzymes that convert calories into fat.3

A new study published earlier this year provided more evidence for coconut oil's growing reputation as a weight management aid. The study compared the health effects of coconut oil with soybean oil. In this study, scientists divided the mice into four groups and fed them each a high-calorie, high-fat diet to promote weight gain. Each of the four diets contained 40 percent fat. One diet used coconut oil, in another group half of the coconut oil was replaced with soybean oil (which primarily contains polyunsaturated fat). The third and fourth diets were the same as the first two but had fructose added. All four diets had the same number of calories, and the mice were fed the same amount of food.

As expected, all of the mice gained weight on the high-calorie diet, but at distinctly different rates. The mice that were on the coconut oil diet gained the least amount, a whopping 25 percent less weight than those on the soybean oil diet. Interestingly, the mice on the soybean oil diet also gained 12 percent more weight than those that ate a fructose diet.

The mice on the soybean oil diet also had larger fat deposits in their bodies, developed fatty livers, and were more likely to develop diabetes and insulin resistance. Also, genes involved mitochondrial dysfunction and cancer were upregulated by the soybean oil diet. Mice on the fructose diet didn't get off easy, either — they had similar issues, but to a less severe degree.4

This study provided further evidence that using coconut oil in place of polyunsaturated vegetable oils, such as soybean oil, can help you better manage your weight and reduce the risks of fatty liver, insulin resistance, diabetes, and even cancer.

And, it's worth noting, the amount of soybean oil the mice ate was similar to what we typically get in our diets.

The bottom line is that we don't need a commercially produced coconut oil-based diet pill to help us manage our weight. Simply replacing the polyunsaturated vegetables oils in our diet with coconut oil can help us better manage our weight and help reduce our risks of liver disease, diabetes, and cancer.



1. Feltrin, KL, et al. Effects of lauric acid on upper gut motility, plasma cholecystokinin and peptide YY, and energy intake are load, but not concentration, dependent in humans. Journal of Physiology 2007;581;767-777.

2. Stubbs, RJ and Harbron, CG. Covert manipulation of the ratio of medium-to long-chain triglycerides in isoenergetically dense diets: effect on food intake in ad libitum feeding men. Int J Obes Relat Metab Disord 1996;20:435-444.

3. Fife, B. The Coconut Ketogenic Diet: Supercharge Your Metabolism, Revitalize Thyroid Function, and Lose Excess Weight. Piccadilly Books, Ltd. 2014.

4. Deol, P, et al. Soybean oil is more obesogenic and diabetogenic than coconut oil and fructose in mouse: potential role for the liver. PLoS ONE 20015;10(7):e0132672.


The Secret Formula for McDonald's French Fries

Does it even contain any real potatoes?You might be surprised


Ever since McDonald's first opened in 1948, the one thing most people rave about is their French fries. As a kid, I loved their French fries better than any other and would go there just for their fries.

McDonalds French fires

 So how do they do it? What makes McDonald's fries better than most everyone else's? It's no accident. McDonald's has spent a lot of time and effort into perfecting their fries, which has evolved over the years.

The original fries were made from russet potatoes, cooked in beef tallow, and finished off with a little salt. Simple, but delicious, primarily because they were cooked in beef tallow that gave the fries their unique savory flavor. However, with the outcry against saturated fats in the late 1980s, most restaurants, including McDonald's, in order to ease customer's fears, replaced the beef tallow with hydrogenated soybean oil. Ignoring the fact that hydrogenated vegetable oils are far worst for your health than beef tallow, the change in oils made a huge impact on the taste and flavor of the fries.

Hydrogenated soybean oil, which is highly processed and tasteless, produced a bland, flavorless French fry that had no resemblance to the delicious fries cooked in beef tallow. So McDonald's began experimenting with the formula adding beef broth and flavorings to mimic the flavor and texture of their original fries. Even the types of oils they used changed over the years. So many changes were made that some people even wondered if they even contained any real potatoes.

Food myth buster, Grant Imahara, was allowed access into the McDonald's French fry processing plant in Idaho, to set the record straight about what ingredients go into McDonald's secret French fry formula.

After visiting the plant and observing the entire process involved in producing the fries, he was able to confirm that potatoes really are the first and primary ingredient in their fries. But don't get too excited just yet. In addition to the potatoes, there are another 18 ingredients! That's right. A total of 19 ingredients goes into making that simple little French fry. And some of these ingredients are not what you might expect.

The formula includes 14 chemicals, one of which is petroleum-based (tertiary butylhydroquinone) and another is a silicone used in silly putty (dimethylpolysiloxane). The fries are made from a mix of russet, Burbank, ranger russet, Umatilla russet, and Shepody potatoes then are peeled cut and blanched before being fired through a cutting cannon at 70 mph. The cut potatoes then pass through a bath of sauces, consisting of a blend of canola oil, corn oil, soybean oil, hydrogenated soybean oil, natural beef flavor, hydrolysed wheat, hydrolysed milk, citric acid, Tertiary butylhydroquinone (TBHQ), and dimethylpolysiloxane. TBHQ is included in the cooking oil as a food preservative and dimethylpolysiloxane is used to stop cooking oils from foaming as the potatoes are deep fried. Finally, dextrose is then sprayed on the fries to ensure they maintain a golden color and sodium acid pyrophosphate is added to stop them from turning gray.

After the potatoes are fried in the factory, they are then flash frozen and shipped off to all the McDonald's restaurants, where they are fried a second time and salted just before serving. The end product is mmmm, mmmm, finger lick'in good...or is that some other fried product with mystery ingredients?


Are We Being Told the Truth About Vaccines?

Whistleblower Reveals CDC Destroyed Documents to Conceal Cover Up of MMR-Autism Link

On July 29, 2015, US Congressman Bill Posey spoke on the floor of the US House quoting whistleblower Dr. Bill Thompson, a senior research scientist at the Center for Disease Control and Prevention (CDC). Dr. Thompson is quoted as stating that the CDC purposely destroyed documents to conceal their tracks when they covered up their own study's findings that MMR vaccine caused a huge risk of autism in black boys when given before 3 years of age. 

syringe with 100 dollar bill in it

The study was published in the Journal of Pediatrics and was used by the pro-vaccine lobby as "proof" that the MMR vaccine did not cause autism. It also provided ammunition to push new vaccine legislation though state governments and restrict parents' rights to make informed decision regarding vaccinations for their children. For example, California's recent move to eliminate all personal exceptions for children from receiving the standard 48 or so vaccinations by age 6 currently mandated.

Below is an excerpt of Congressman Posey's address:                               

 I rise today on matters of research and scientific integrity. To begin with, I am absolutely, resolutely pro-vaccine. Advancements in medical immunization have saved countless lives and greatly benefitted public health. 

That being said, it's troubling to me that in a recent Senate hearing on childhood vaccinations, it was never mentioned that our government has paid out over three billion dollars through a vaccine injury compensation program for children who have been injured by vaccinations. Regardless of the subject matter, parents making decisions about their children's health deserve to have the best information available to them. They should be able to count on federal agencies to tell them the truth. For these reasons I bring the following matter to the House floor.

In August 2014, Dr. William Thompson, a senior scientist at the Centers for Disease Control and Prevention worked with a whistleblower attorney to provide my office with documents related to a 2004 CDC study that examined the possibility of a relationship between mumps, measles, rubella (MMR) vaccines and autism. In a statement released in August 2014, Dr. Thompson stated, "I regret that my co-authors and I omitted," OMITTED, "statistically significant information in our 2004 article published in the Journal of Pediatrics."

Mr. Speaker, I respectfully request the following excerpts from the statement written by Dr. Thompson be entered into the record. Now quoting Dr. Thompson: 

"My primary job duties while working in the immunization safety branch from 2000-06 were to lead or co-lead three major vaccine safety studies. The MADDSP MMR Autism Cases controlled study was being carried out in response to the Wakefield Lancet study that suggested an association between the MMR vaccine and an autism-like health outcome. There were several major concerns among scientists and consumers advocates outside the CDC in the fall of 2000 regarding the execution of the Verstraeten study. One of the important goals that was determined up front in the spring of '01 before any of these studies started was to have all three protocols vetted outside the CDC prior to start of the analyses so that consumer advocates could not claim that we were presenting analyses that suited our own goals and biases. We hypothesized that if we found statistically significant effects at either 18 or 36 month thresholds, we would conclude that vaccinating children early with MMR vaccine could lead to autism-like characteristics or features.

"We all met and finalized the study protocol and analysis plan. The goal was to not deviate from the analysis plan to avoid the debacle that occurred with the Verstraeten thimerosal study published in Pediatrics in '03. At the September 5th meeting we discussed in detail how to code race for both the sample and the birth certificate sample. At the bottom of Table 7, it also shows that for the non-birth-certificate sample, the adjusted race effect statistical significance was huge. All the authors and I met and decided sometime between August and September '02 not to report any race effects for the paper. Sometime soon after the meeting we decided to exclude reporting any race effects, the co-authors scheduled a meeting to destroy documents related to the study. The remaining four co-authors all met and brought a big garbage can into the meeting room and reviewed and went through all the hardcopy documents that we had thought we should discard and put them in a huge garbage can. However, because I assumed it was illegal and would violate both FOIA and DOJ requests, I kept hard copies of all documents in my office, and I retained all associated computer files. 

"I believe we intentionally withheld controversial findings from the final draft of the Pediatrics paper."

Mr. Speaker, I believe it's our duty to insure that the documents Dr. Thompson provided are not ignored. Therefore, I will provide them to members of Congress and the House committees upon request. Considering the nature of the whistleblower's documents, as well as the involvement of the CDC, a hearing and a thorough investigation is warranted. So I ask, Mr. Speaker, I beg, I implore my colleagues on the appropriations committees to please, please take such action. Thank you, Mr. Speaker.


 The vaccine makers and their allies in government, are working to take away our right to be informed about the dangers of vaccinations and to force us to be vaccinated, even against our will. If this is of concern to you, I urge you to take action and let your voice be heard.

Send an email to Congressman Posey AND your own Congressperson.  The email does not have to be long, all it needs to say is something like this:

"I support Rep. Bill Posey and thank him for his testimony on July 29, 2015 regarding the CDC's intentional destruction of documents. I am grateful William Thompson, MD retained hard copies documenting the truth and as a US citizen I am requesting a full investigation into this issue."

Remember to include your name and address.



Send EMAILS of support directly to Bill Posey's assistant Patricia Febro at Patricia.Febro@mail.house.gov





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